Information de reference pour ce titreAccession Number: | 00124509-201503000-00022.
|
Author: | Ghignone, Erica MD *; Rosenthal, Lisa MD *; Lloyd, Robert Brett MD, PhD *; Mouli, Samdeep MD +; Dinwiddie, Stephen MD *
|
Institution: | From the Departments of *Psychiatry and Behavioral Sciences, and +Radiology, Feinberg School of Medicine, Northwestern University, Chicago, IL.
|
Title: | |
Source: | Journal of ECT. 31(1):e14-e16, March 2015.
|
Abstract: | We report on a 30-year-old woman diagnosed with moyamoya syndrome resulting from sickle cell disease who developed catatonia and was successfully treated with electroconvulsive therapy (ECT). Neuroimaging revealed severe tandem narrowing of the left internal carotid artery with diminished cerebral blood flow, moderate narrowing of the right supraclinoid aspect of the right internal carotid artery, and associated numerous lenticulostriate collaterals bilaterally, consistent with moyamoya. The patient presented with mutism; posturing; immobility; stupor; withdrawal; refusal to eat, drink, or speak; and staring, supporting a diagnosis of catatonia. It initially responded to a lorazepam challenge; however, a complicated hospital course and deterioration of the patient's condition, including septic shock, delirium, and continued catatonic symptoms, led to the pursuit of ECT to treat her symptoms. We discuss the risks involved with the administration of ECT in a patient with fragile cerebral vasculature and the successful treatment of catatonia in this patient without resultant stroke or cerebral hemorrhage.
Copyright (C) 2015 Wolters Kluwer Health, Inc. All rights reserved
|
Author Keywords: | electroconvulsive therapy; moyamoya syndrome; sickle cell disease; catatonia.
|
References: | 1. Takeuchi K, Shimizu K. Hypoplasia of the bilateral internal carotid arteries. Brain Nerve. 1957; 9: 37-43.
2. Suzuki J, Takaku A. Cerebrovascular "moyamoya" disease: disease showing abnormal net-like vessels in base of brain. Arch Neurol. 1969; 20: 288-299.
3. Scott RM, Smith ER. Moyamoya disease and moyamoya syndrome. N Engl J Med. 2009; 360: 1226-1237.
4. Lubman DI, Pantelis C, Desmond P, et al. Moyamoya disease in a patient with schizophrenia. J Int Neuropsychol Soc. 2003; 9: 806-810.
5. Daniels J. Catatonia: clinical aspects and neurobiological correlates. J Neuropsychiatr Clin Neurosci. 2009; 21: 371-380.
6. Fink M. Catatonia: a syndrome appears, disappears, and is rediscovered. Can J Psychiatry. 2009; 54: 437-445.
7. Unal A. Effective treatment of catatonia by combination of benzodiazepine and electroconvulsive therapy. J ECT. 2013; 29: 206-209.
8. Yamashita M, Oka K, Tanaka K. Histopathology of the brain vascular network in moyamoya disease. Stroke. 1983; 14: 50-58.
9. Gitig A, Merino JG, Hoffman L, et al. Moyamoya disease presenting as atypical post-ictal delirium after ECT in a woman with clinically silent putaminal infarcts. Einstein Q J Biol Med. 2000; 17: 190-193.
|
Language: | English.
|
Document Type: | Case Reports.
|
Journal Subset: | Nursing. Clinical Medicine. Health Professions.
|
ISSN: | 1095-0680
|
NLM Journal Code: | cxp, 9808943
|
DOI Number: | https://dx.doi.org/10.1097/YCT.0...- ouverture dans une nouvelle fenêtre
|
Annotation(s) | |