comparison of the pharmacokinetics, acid suppression and efficacy of proton pump inhibitors
the clinical influence of Helicobacter pylori in effective acid suppression—implications for the treatment of gastro-oesophageal reflux disease
treatment of Helicobacter pylori infection with ranitidine bismuth citrate- or proton pump inhibitor-based triple therapies
Non-absorbable antibiotics for managing intestinal gas production and gas-related symptoms
Enhanced bioavailability of azathioprine compared to 6-mercaptopurine therapy in inflammatory bowel disease: correlation with treatment efficacy
Randomized comparison of unfractionated heparin with corticosteroids in severe active inflammatory bowel disease
Chemotactic properties of ICAM-1 and PECAM-1 on neutrophil granulocytes in ulcerative colitis: effects of prednisolone and mesalazine
Topical and intravenous administration of trefoil factors protect the gastric mucosa from ethanol-induced injury in the rat
Cisapride provides symptomatic relief in functional dyspepsia associated with gastric myoelectrical abnormality
Helicobacter pylori infection influences nocturnal gastric acid breakthrough
Pharmacodynamics and kinetics of omeprazole MUPS 20 mg and pantoprazole 40 mg during repeated oral administration in Helicobacter pylori-negative subjects
Equally high efficacy of 4, 7 and 10-day triple therapies to eradicate Helicobacter pylori infection in patients with ulcer disease
Ulcer-healing drugs are required after eradication of Helicobacter pylori in patients with gastric ulcer but not duodenal ulcer haemorrhage
Pantoprazole versus one-week Helicobacter pylori eradication therapy for the prevention of acute NSAID-related gastroduodenal damage in elderly subjects
Rabeprazole, amoxycillin and low- or high-dose clarithromycin for cure of Helicobacter pylori infection
Cetraxate, a mucosal protective agent, combined with omeprazole, amoxycillin, and clarithromycin increases the eradication rate of Helicobacter pylori in smokers
Nitric oxide formation in the dog sphincter of Oddi from nitric oxide donors as measured with in vivo micro-dialysis
Lifestyle measures and reflux
OAM for cure of Helicobacter pylori infection
Acid suppression and upper GI bleeding
Helicobacter pylori infection is a protective factor for bleeding gastric ulcers but not for bleeding duodenal ulcers in NSAID users
Reply —Helicobacter pylori infection is a protective factor for bleeding gastric ulcers but not for bleeding duodenal ulcers in NSAID users