Involvement of the Hypothalamic-Pituitary-Adrenal/Gonadal Axis and the Peripheral Nervous System in Rheumatoid Arthritis : Viewpoint Based on a Systemic Pathogenetic Role
Treatment of Polyarteritis Nodosa, Microscopic Polyangiitis, and Churg-Strauss Syndrome: Where Do We Stand?
Winners of the 2000 American College of Rheumatology Slide Competition
A Randomized Two-Year Study of the Effects of Dynamic Strength Training on Muscle Strength, Disease Activity, Functional Capacity, and Bone Mineral Density in Early Rheumatoid Arthritis
Increased Mortality in Adults With a History of Juvenile Rheumatoid Arthritis : A Population-Based Study
Indirect Medical Costs in Early Rheumatoid Arthritis : Composition of and Changes in Indirect Costs Within the First Three Years of Disease
Influence of Shared Epitope-Negative HLA-DRB1 Alleles on Genetic Susceptibility to Rheumatoid Arthritis
Lymph Draining From Foot Joints in Rheumatoid Arthritis Provides Insight Into Local Cytokine and Chemokine Production and Transport to Lymph Nodes
Levels of Interleukin-18 and Its Binding Inhibitors in the Blood Circulation of Patients With Adult-Onset Still's Disease
Shift Toward T Helper 1 Cytokines by Type II Collagen-Reactive T Cells in Patients With Rheumatoid Arthritis
Infection Efficiency of Type 5 Adenoviral Vectors in Synovial Tissue Can Be Enhanced With a Type 16 Fiber
Osteopontin : An Intrinsic Inhibitor of Inflammation in Cartilage
Matrix Metalloproteinase and Proinflammatory Cytokine Production by Chondrocytes of Human Osteoarthritic Cartilage : Associations With Degenerative Changes
Peroxisome Proliferator-Activated Receptor γ Activators Inhibit Interleukin-1β-Induced Nitric Oxide and Matrix Metalloproteinase 13 Production in Human Chondrocytes
Cyclic Tensile Strain Suppresses Catabolic Effects of Interleukin-1β in Fibrochondrocytes From the Temporomandibular Joint
The Fcγ Receptor IIIA-158F Allele Is a Major Risk Factor for the Development of Lupus Nephritis Among Caucasians but Not Non-Caucasians
Immunoglobulin Vκ Light Chain Gene Analysis in Patients With Sjögren's Syndrome
Polymorphisms of the Ro52 Gene Associated With Anti-Ro 52-kd Autoantibodies in Patients With Primary Sjögren's Syndrome
Long-Term Outcome of Mothers of Children With Isolated Heart Block in Finland
The Disability Index of the Health Assessment Questionnaire Is a Predictor and Correlate of Outcome in the High-Dose Versus Low-Dose Penicillamine in Systemic Sclerosis Trial
Cytochrome P2 Polymorphisms and Susceptibility to Scleroderma Following Exposure to Organic Solvents
Long-Term Followup of Polyarteritis Nodosa, Microscopic Polyangiitis, and Churg-Strauss Syndrome : Analysis of Four Prospective Trials Including 278 Patients
Invasiveness of Synovial Fibroblasts Is Regulated by p53 in the SCID Mouse In Vivo Model of Cartilage Invasion
Variations in Susceptibility to Proteoglycan-Induced Arthritis and Spondylitis Among C3H Substrains of Mice : Evidence of Genetically Acquired Resistance to Autoimmune Disease
Elevated Proapoptotic Bax and Caspase 3 Activation in the NOD.scid Model of Sjögren's Syndrome
Cysteine Protease Activity Is Up-Regulated in Inflamed Ankle Joints of Rats With Adjuvant-Induced Arthritis and Decreases With In Vivo Administration of a Vinyl Sulfone Cysteine Protease Inhibitor
Activation of a Fibroblast-Specific Enhancer of the Proα2(I) Collagen Gene in Tight-Skin Mice
Development of the Tight-Skin Phenotype in Immune-Deficient Mice
Prolonged remission without treatment after autologous stem cell transplantation for refractory childhood systemic lupus erythematosus
Up-regulated expression of transforming growth factor β receptors in dermal fibroblasts in skin sections from patients with localized scleroderma
Updating the American College of Rheumatology preliminary classification criteria for systemic sclerosis: addition of severe nailfold capillaroscopy abnormalities markedly increases the sensitivity for limited scleroderma
Possible role of antiidiotypes in the loss of anti-topoisomerase I antibodies: comment on the article by Kuwana et al
Possible role of antiidiotypes in the loss of anti-topoisomerase I antibodies: comment on the article by Kuwana et al