Editorial
On the weight of the brain and its component parts in the insane. By J. Crichton-Browne, MD, FRSE, Lord Chancellor's Visitor. Brain 1879: 1; 514–518 and 1879: 2; 42–67.
From hodology to function
Mutations in SUCLA2: a tandem ride back to the Krebs cycle
Stimulating language: insights from TMS
Does the left inferior longitudinal fasciculus play a role in language? A brain stimulation study
Association fibre pathways of the brain: parallel observations from diffusion spectrum imaging and autoradiography
Clinical findings and white matter abnormalities seen on diffusion tensor imaging in adolescents with very low birth weight
Perinatal risk factors altering regional brain structure in the preterm infant
Volume, neuron density and total neuron number in five subcortical regions in schizophrenia
Altered orbitofrontal sulcogyral pattern in schizophrenia
Focal atrophy in dementia with Lewy bodies on MRI: a distinct pattern from Alzheimer's disease
The topography of grey matter involvement in early and late onset Alzheimer's disease
Profiles of neuropsychological impairment in autopsy-defined Alzheimer's disease and cerebrovascular disease
Auditory cortical activity in amnestic mild cognitive impairment: relationship to subtype and conversion to dementia
Loss of insight in frontotemporal dementia, corticobasal degeneration and progressive supranuclear palsy
Interictal habituation deficit of the nociceptive blink reflex: an endophenotypic marker for presymptomatic migraine?
Impaired filtering of behaviourally irrelevant visual information in dyslexia
Specific influences of cerebellar dysfunctions on gait
Long-term consequences of human alpha-synuclein overexpression in the primate ventral midbrain
Atypical parkinsonism in Guadeloupe: a common risk factor for two closely related phenotypes?
The phenotypic spectrum of rapid-onset dystonia–parkinsonism (RDP) and mutations in the ATP1A3 gene
VHL mutation analysis in patients with isolated central nervous system haemangioblastoma
The spectrum of SCN1A-related infantile epileptic encephalopathies
Mitochondrial encephalomyopathy with elevated methylmalonic acid is caused by SUCLA2 mutations
SUCLA2 mutations are associated with mild methylmalonic aciduria, Leigh-like encephalomyopathy, dystonia and deafness
FIBRE PATHWAYS OF THE BRAIN
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