Genetic studies of syndromes with severe periodontitis and palmoplantar hyperkeratosis
Evidence for genetic control of changes in f-actin polymerization caused by pathogenic microorganisms: in vitro assessment using gingival fibroblasts from human twins
Lipopolysaccharide dependent cellular activation
Molecular factors associated with compartmentalization of gingival immune responses and transepithelial neutrophil migration
Immunoregulatory roles of adhesive interactions between lymphocytes and gingival fibroblasts
Molecular and cellular mechanisms for periodontal diseases: role of Th1 and Th2 type cytokines in induction of mucosal inflammation
Porphyromonas gingivalis proteinases as virulence factors in the development of periodontitis
Are bacterial proteases important virulence factors?
Analysis of the protease and adhesin domains of the PrpRI ofPorphyromonas gingivalis
Multiple colonization defects in a cysteine protease mutant ofPorphyromonas gingivalis
The reversal line may be a key modulator of osteoblast function: observations from an alveolar bone wound-healing model
Markers of bone and cementum formation accumulate in tissues regenerated in periodontal defects treated with expanded polytetrafluoroethylene membranes
Fibroblast heterogeneity in the periodontium and other tissues
Underlying mechanisms at the bone-surface interface during regeneration
Biological effects of drug-loaded biodegradable membranes for guided bone regeneration
Crevicular fluid and serum IgG subclasses and corresponding mRNA expressing plasma cells in periodontitis lesions
The effect of age on the expression of mineralized tissue progenitors in the periodontium - the effect of bFGF
Tetracycline inhibits Prophyromonas gingivalis lipopolysaccharide-induced lesions in vivo and TNFα processing in vitro
Interleukin-10 promotes anti-collagen antibody production in type I diabetic peripheral B lymphocytes
Bacterial metabolite mediated differential human PMN gene expression
Interactions between periodontopathogenic bacteria and cytokines
Incomplete penetrance, white space-black space, disease perspective: infectious disease vs. molecular medicine