AbstractBackground and Purpose—
Insular cortex ischemia is proposed to mediate a sympathetic stimulus that leads to acute hyperglycemia after stroke.Methods—
We retrospectively analyzed insular perfusion on perfusion CT (median 180 minutes after onset) in 35 patients.Results—
We found no association of hypoperfusion (relative cerebral blood flow <0.51) with early (<6 hours) or delayed (<72 hours) hyperglycemia, or hemispheric lateralization.Conclusions—
Insular cortex hypoperfusion <6 hours after stroke onset was not associated with hyperglycemia.