Developmental bisphenol A (BPA) exposure leads to sex-specific modification of hepatic gene expression and epigenome at birth that may exacerbate high-fat diet-induced hepatic steatosis
Honokiol activates the LKB1–AMPK signaling pathway and attenuates the lipid accumulation in hepatocytes
The imperatorin derivative OW1, a new vasoactive compound, inhibits VSMC proliferation and extracellular matrix hyperplasia
Characterizing the mechanism of thiazolidinedione-induced hepatotoxicity: An in vitro model in mitochondria
Developmental exposure to 2,3,7,8-tetrachlorodibenzo- p -dioxin alters DNA methyltransferase ( dnmt ) expression in zebrafish ( Danio rerio )
Paradoxically, iron overload does not potentiate doxorubicin-induced cardiotoxicity in vitro in cardiomyocytes and in vivo in mice
Combination effects of AHR agonists and Wnt/β-catenin modulators in zebrafish embryos: Implications for physiological and toxicological AHR functions
Potential of extracellular microRNAs as biomarkers of acetaminophen toxicity in children
Sex-related differences in murine hepatic transcriptional and proteomic responses to TCDD
Quaternary and tertiary aldoxime antidotes for organophosphate exposure in a zebrafish model system
A rat model of nerve agent exposure applicable to the pediatric population: The anticonvulsant efficacies of atropine and GluK1 antagonists
Metformin inhibits 7,12-dimethylbenz[a]anthracene-induced breast carcinogenesis and adduct formation in human breast cells by inhibiting the cytochrome P4501A1/aryl hydrocarbon receptor signaling pathway
A novel synthetic derivative of melatonin, 5-hydroxy-2′-isobutyl-streptochlorin (HIS), inhibits inflammatory responses via regulation of TRIF-dependent signaling and inflammasome activation
Regulation of ozone-induced lung inflammation and injury by the β-galactoside-binding lectin galectin-3
In vitro biotransformation of tris(2-butoxyethyl) phosphate (TBOEP) in human liver and serum
Sodium meta-arsenite prevents the development of autoimmune diabetes in NOD mice
Predicting chemically-induced skin reactions. Part I: QSAR models of skin sensitization and their application to identify potentially hazardous compounds
Predicting chemically-induced skin reactions. Part II: QSAR models of skin permeability and the relationships between skin permeability and skin sensitization