TARGETED AND SUSTAINED EFFECT OF NO BY S-NITROSATION OF CYSTEINE RESIDUES IN ARTERIES EXHIBITING ENDOTHELIAL DYSFUNCTION
ENDOTHELIUM-DEPENDENT NO-MEDIATED RELAXATIONS TO CONCORD GRAPE JUICE IS MEDIATED THROUGH THE REDOX-SENSITIVE PI3-KINASE/AKT-DEPENDENT PHOSPHORYLATION OF ENDOTHELIAL NO SYNTHASE IN THE PORCINE CORONARY ARTERY AND RAT AORTA
ADRENOCEPTORS ON ENDOTHELIUM REVISITED
MECHANISMS INVOLVED IN THE RELAXATION OF RAT BASILAR ARTERY INDUCED BY THE SOLUBLE GUANYLYL CYCLASE STIMULATOR BAY 41-2272.
DYNAMIC ASSOCIATION OF SOLUBLE GUANYLYL CYCLASE AND PROTEIN KINASE A WITH CAVEOLIN-1 IN RAT AORTIC ENDOTHELIUM MEDIATING VASODILATION
A MODEL FOR HYPERACUTE HYPOTENSION AND SHOCK: NITRIC OXIDE OR NO NITRIC OXIDE?
RENOVASCULAR HYPERTENSION AND ACUTE PRESSURE ELEVATION SELECTIVELY IMPAIRS NO-MEDIATED VASODILATION IN RAT SMALL ARTERIES
FLOW AUGMENTS THE CONTRACTIONS TO PHENYLEPHRINE IN THE RABBIT CAROTID ARTERY BY REDUCING THE RELEASE OF ENDOTHELIUM-DERIVED NO
BLOOD PRESSURE IN MICE WITH A TARGETED DELETION OF THE SOLUBLE GUANYLYL CYCLASE ALPHA1 SUBUNIT
THE ROLE OF PERTUSSIS TOXIN SENSITIVE PATHWAYS IN EDHF MEDIATED RELAXATION IN PORCINE CORONARY ARTERIES
FUNCTIONAL EVALUATION OF EPOXYEICOSATRIENOIC ACIDS AS ENDOTHELIUM- DERIVED HYPERPOLARIZING FACTOR IN RAT MESENTERIC ARTERIAL BED
EDHF- BUT NOT NO-MEDIATED RELAXATION IS ABOLISHED IN THE ISOLATED MESENTERIC ARTERIES OF HYPERTENSIVE RATS CHRONICALLY TREATED WITH ANGIOTENSIN II
THE ROLE OF ENDOTHELIAL CONNEXIN 40 AND CONNEXIN 43 mRNA EXPRESSION IN RELATION TO HYPERHOMOCYSTEINEMIA
SMALLER WINDOWS IN INTERNAL ELASTIC LAMINA, FEWER ROUTES FOR MYOENDOTHELIAL JUNCTIONS, LESS EDHF RESPONSE.
ENDOTHELIUM-DEPENDENT RESPONSES OF CAROTID, FEMORAL AND MESENTERIC ARTERIES OF STREPTOZOTOCIN-INDUCED DIABETIC RATS
IMPAIRED EDHF-DILATION CORRELATED WITH INCREASED MYOGENIC CONSTRICTION IN MESENTERIC RESISTANCE ARTERIES DURING PROGRESSION OF HEART FAILURE
IMPAIRED EDHF RESPONSES IN RENAL ARTERIES FROM THE OBESE ZUCKER RAT: INVOLVEMENT OF GAP JUNCTIONS AND CONNEXINS
REGIONAL DIFFERENCES IN ENDOTHELIAL HYPERPOLARIZATION AND ENDOTHELIUM-DERIVED HYPERPOLARIZING FACTOR IMPAIRMENT IN DIABETES MELLITUS
RALOXIFENE ATTENUATES ENDOTHELIAL DYSFUNCTION INDUCED BY OXIDIZED LOW-DENSITY LIPOPROTEIN
CALCIUM HANDLING IN AORTIC SMOOTH MUSCLE CELLS OF APOLIPOPROTEIN E-DEFICIENT MICE TRANSFECTED WITH PARAOXONASE-1
ENDOTHELIUM-DEPENDENT CONTRACTIONS IN THE SHR AORTA: THE JANUS FACE OF PROSTACYCLIN
THE LIPID PEROXIDATION INHIBITOR BUTYLATED HYDROXYTOLUENE (BHT) ANNULS BRADYKININ INDUCED VASODILATION RESISTANT TO NOS & COX INHIBITORS IN THE RAT MESENTERIC AND CORONARY BEDS.
ROSUVASTATIN'S PROTECTION AGAINST THE DEVELOPMENT OF NITRATE TOLERANCE IN WILD-TYPE AND eNOS−/− MICE SEEMS TO IMPLICATE THE NAD(P)H OXIDASE PATHWAY.
ACETYLCHOLINE AND SODIUM NITROPRUSSIDE CAUSE LONG-TERM INHIBITION OF EDCF-MEDIATED CONTRACTIONS
EFFECTS OF ASCORBATE ON ENDOTHELIUM-DERIVED HYPERPOLARIZING FACTOR (EDHF)-MEDIATED VASODILATATION IN THE PERFUSED RAT MESENTERY AND HINDLIMB
CHARACTERIZATION OF PURINERGIC P2Y RECEPTORS ON ENDOTHELIAL CELLS OF THE MOUSE AORTA
NEUROPEPTIDE Y2 RECEPTORS ARE INVOLVED IN ENHANCED NEUROGENIC VASOCONSTRICTION IN SPONTANEOUSLY HYPERTENSIVE RATS
STUDY ON THE IDENTITY AND THE CHARACTERISTICS OF THE RETINAL RELAXING FACTOR (RRF)
APOLIPOPROTEIN E-DEFICIENT MICE SHOW ENHANCED CALCIUM-DEPENDENT RELAXATION TO ACETYLCHOLINE BEFORE DEVELOPMENT OF ATHEROSCLEROTIC LESIONS.
RHO-KINASE AND CALCIUM SIGNALING IN ALPHA-1 ADRENERGIC CONTRACTION OF RESISTANCE ARTERIES OF LYON GENETICALLY HYPERTENSIVE RATS
EXERCISE AND VASORELAXING EFFECTS OF CARBON MONOXIDE-RELEASING MOLECULES IN HYPERTENSIVE RATS.
THE DECREASED PRODUCTION OF COLLAGEN TYPE III IN CULTURED SMOOTH MUSCLE CELLS FROM VARICOSE VEIN PATIENTS IS DUE TO A DEGRADATION BY MMPS: POSSIBLE IMPLICATION OF MMP-3
VASCULAR SMOOTH MUSCLE HYPERPOLARIZATION BY CANNABINOIDS IN GASTRIC AND MESENTERIC ARTERIES INVOLVES STIMULATION OF VR1 RECEPTORS ON PERIVASCULAR CGRP CONTAINING NERVES
REDUCED EDHF RESPONSES AND CONNEXIN ACTIVITY IN MESENTERIC ARTERIES FROM THE OBESE ZUCKER RAT
CONTRIBUTION OF NITRIC OXIDE AND OXIDATIVE STRESS TO IMPAIRED CORONARY VASODILATATION AFTER MYOCARDIAL INFARCTION
HYPOXIA-INDUCED ENDOTHELIAL DYSFUNCTION IN HUMAN UMBILICAL VEIN ENDOTHELIAL CELLS
PROGESTINS DECREASE THE EXPRESSION OF ENDOTHELIAL NO SYNTHASE AND THE FORMATION OF NO THROUGH ACTIVATION OF THE GLUCOCORTICOID RECEPTOR
SYNERGISM OF K+- AND HYPEROSMOLARITY-INDUCED RELAXATIONS ON RAT SKELETAL MUSCLE ARTERIOLES.
ENDOTHELIAL BETA3-ADRENOCEPTORS MEDIATE THE NO-DEPENDENT ANGIOGENESIS AND VASORELAXATION OF CORONARY MICROVESSELS IN RESPONSE TO THE THIRD-GENERATION BETA-BLOCKER, NEBIVOLOL
MECHANISMS UNDERLYING THE BIPHASIC NEUROGENIC VASODILATATION IN THE BOVINE INTRAOCULAR LONG POSTERIOR CILIARY ARTERY
CA2+-DEPENDENT K+-CHANNELS AND ENDOTHELIUM DEPENDENT HYPERPOLARIZATION IN RAT ISOLATED CEREBRAL ARTERIES; POSSIBLE REGULATION BY NO?
S-NITROSO-N-ACETYLCYSTEINE PREVENTS LEFT VENTRICULAR HYPERTROPHY BUT NOT INCREASED ARTERIAL PRESSURE IN HYPERCHOLESTEROLEMIC LDLR−/− MICE
CELLULAR MECHANISMS UNDERLYING CUTANEOUS PRESSURE-INDUCED VASODILATION: IN VIVO INVOLVEMENT OF POTASSIUM CHANNELS
CHRONIC ORAL ADMINISTRATION OF RED WINE POLYPHENOLS PREVENTS THE ANGIOTENSIN II-INDUCED HYPERTENSION AND ENDOTHELIAL DYSFUNCTION BY INHIBITING THE VASCULAR EXPRESSION OF NADPH OXIDASE
ACE INHIBITORS ENHANCE ENDOTHELIAL CYCLOOXYGENASE-2 (COX-2) EXPRESSION VIA JNK-DEPENDENT ACE-SIGNALLING
NAD(P)H OXIDASE-DERIVED SUPEROXIDE ANIONS DO NOT CONTRIBUTE TO ENDOTHELIUM-DERIVED HYPERPOLARIZING FACTOR/ H2O2 RESPONSES IN MICE
POTASSIUM IS A POTENTIAL EDHF IN HUMAN CORONARY MICROARTERIES.
CEREBRAL VASCULAR MITOCHONDRIAL EFFICIENCY IS INCREASED BY ESTROGEN TREATMENT
IMPAIRED HISTAMINE-INDUCED RELAXATION IN MESENTERIC ARTERY OF DOCA-SALT HYPERTENSIVE RATS INVOLVES CHANGES ON MECHANISMS OF NO-INDEPENDENT HYPERPOLARIZATION
CALCIUM HANDLING AND ISOMETRIC FORCE DEVELOPMENT IN AORTIC SMOOTH MUSCLE CELLS OF APOLIPOPROTEIN E-DEFICIENT MICE
IMPAIRED SAPHENOUS ARTERY REACTIVITY IN THE SETTING OF HINDLIMB ISCHEMIA
CILOSTAZOL IMPROVES EDHF-TYPE RELAXATION IN THE MESENTERIC ARTERY OF STREPTOZOTOCIN-INDUCED DIABETIC RATS
BIPHASIC RESPONSES TO PROSTACYCLIN AND ACETYLCHOLINE IN ISOLATED PERFUSED MOUSE KIDNEYS: INVOLVEMENT OF TP RECEPTORS.
BALANCE BETWEEN EDHF AND NO RELEASED FROM RENAL ARTERIES OF SHR/NDMCR-CP RATS, A MODEL FOR THE METABOLIC SYNDROME
ENDOTHELIAL DYSFUNCTION IN THE TYPE TWO DIABETIC db/db MOUSE IS ASSOCIATED WITH AN UPREGULATION OF A SARCOLEMMAL MEMBRANE ASSOCIATED PROTEIN, SLMAP
CIRCULATING ENDOTHELIAL MICROPARTICLES IMPAIR ENDOTHELIAL FUNCTION IN PATIENTS WITH END STAGE RENAL FAILURE
ENDOTHELIUM-DERIVED HYDROGEN PEROXIDE ACCOUNTS FOR THE ENHANCING EFFECT OF AN ANGIOTENSIN-CONVERTING ENZYME INHIBITOR ON ENDOTHELIUM-DERIVED HYPERPOLARIZING FACTOR-MEDIATED RESPONSES IN MICE
EARLY ENDOTHELIAL DYSFUNCTION SEVERELY IMPAIRS SKIN BLOOD FLOW RESPONSE TO LOCAL PRESSURE APPLICATION IN STREPTOZOTOCIN-DIABETIC MICE
AN EXAMINATION OF THE EFFECTS OF CANNABINOID AGONISTS ON ATP RELEASE FROM SYMPATHETIC NERVES
ROLE OF THE JNK AND HIF1-ALPHA IN THE INHIBITION OF VEGF EXPRESSION BY GREEN TEA CATECHINS IN VASCULAR SMOOTH MUSCLE CELLS
EFFECTS OF INDUCING TONE WITH ENDOTHELIN-1 ON NERVE-MEDIATED CONTRACTILE RESPONSES IN THE RAT MESENTERIC VASCULAR BED
CONNEXIN EXPRESSION AND ENDOTHELIUM-DEPENDENT VASODILATOR RESPONSES IN OMENTAL ARTERIES FROM NORMOTENSIVE PREGNANT WOMEN.
MODULATION OF CALCIUM HOMEOSTASIS BY NO IN PORCINE ENDOTHELIAL CELLS
PIVOTAL ROLE OF CU,ZN-SOD IN ENDOTHELIUM-DEPENDENT HYPERPOLARIZATION IN MICE AND HUMANS
RHO KINASE PLAYS AN IMPORTANT ROLE IN THE POSTNATAL CHANGE IN NITRIC OXIDE AND cGMP-MEDIATED RELAXATION OF OVINE CORONARY ARTERIES
EFFECT OF THIAZOLIDINEDIONES ON ADENOSINE TRANSPORT IN VASCULAR SMOOTH MUSCLE CELLS
GENDER-DEPENDENT BASAL VASODILATOR ACTIVITY OF ENDOTHELIAL CELLS IN APOLIPOPROTEIN E-DEFICIENT MICE
INFLUENCE OF DIABETES MELLITUS, HYPERCHOLESTEROLEMIA, AND THEIR COMBINATION ON EDHF-MEDIATED RELAXATIONS IN MICE
NON-GENOMIC VASCULAR EFFECT OF 17β-ESTRADIOL INVOLVES ACTIVATION OF ADENYLYL CYCLASE AND PROTEIN KINASE G IN THE RAT MESENTERIC ARTERY
EDHF-MEDIATED RELAXATION IN RABBIT CAROTID ARTERY IS DEPENDENT ON LUMINAL FLOW
RETINAL TISSUE PRODUCES ACUTE AND BIPHASIC RELAXATIONS IN ISOLATED BOVINE RETINAL ARTERIES
INDUCTION OF CYTOCHROME P450 2C BY CYCLIC STRAIN ENHANCES COX-2 EXPRESSION AND ENDOTHELIAL TUBE FORMATION
KAEMPFEROL ENHANCES ENDOTHELIUM-INDEPENDENT RELAXATION: THE ROLE OF cAMP PATHWAY AND K+ CHANNELS
CYTOCHROME P450 2C-DERIVED EPOXYEICOSATRIENOIC ACIDS (EETS) REGULATE Ca2+ SIGNALLING AND HYPERPOLARIZATION IN ENDOTHELIAL CELLS.
A METHOD FOR RAPID SEPARATION OF ENDOTHELIUM AND SMOOTH MUSCLE RNA FROM RAT AORTA: ITS APPLICATION TO ANALYSIS OF NA,K-ATPASE ISOFORM EXPRESSION IN NATIVE ENDOTHELIAL CELLS.
ANTIOXIDANT ACTIVITY OF 5-MTHF AND BH4 CAN PRESERVE EDHF-TYPE RESPONSES BY MODULATING ELECTROTONIC SIGNALLING
IMPAIRED RENAL MYOGENIC CONSTRICTION IS ACCOMPANIED BY PARALLEL PROSTAGLANDIN-DEPENDENT ENDOTHELIAL DYSFUNCTION AND INCREASED NITRIC OXIDE-MEDIATED DILATION IN A GENETIC MODEL OF HYPERTENSION-INDUCED RENAL DISEASE.
GONADAL HORMONES MODULATE LPS-INDUCED INFLAMMATORY MARKERS IN RAT CEREBRAL BLOOD VESSELS
GUANYLYL CYCLASE ACTIVATORS EVOKE NITRIC OXIDE RELEASE IN THE INTACT RAT SUPERIOR MESENTERIC ARTERY
EPOXYEICOSATRIENOIC ACID SYNTHESIS CONTRIBUTES TO ENDOTHELIUM-DEPENDENT HYPERPOLARIZATION IN THIRD-ORDER MESENTERIC RESISTANCE ARTERIES OF eNOS-DEFICIENT MICE
ENDOTHELIUM CALCIUM SIGNAL AND EDHF-DEPENDENT RELAXATION IN DIFFERENT MODELS OF HYPERTENSION
SOME CLINICAL AND HEMODYNAMIC ASPECTS OF THE USE OF LASER THERAPY IN STABLE ANGINA